By John Barrett
- Format: Board Book
- Publication Date: 10/12/1984
- Pages: 28
- Reading point: Age 2 and Up
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Additional info for Big Bird's Mother Goose
Extrarenal manifestations are not observed. The pathogenesis of nephronophthisis is unknown. Fanconi et al. (1951) discussed a "constitutional inferiority of the renal tissue" with tubular destruction and secondary sclerosis. Other authors suggested the possibility of a nephrotoxic substance which leads to early tubular dysfunction and progressive renal damage. This hypothesis is favored by the well-known similarity of nephronophthisis to Balkan nephropathy. The latter is thought to be an environmental disease and caused by an as yet unidentified exogenous toxin (Hall and Dammin 1978).
Cases with recessive inheritance usually present with their firsts;rmptoms during childhood, whereas the dominant pattern has an onset in the third and fourth decade of' life. However, the duration of the renal disease does not appear to be different. between these two groups (Gardener 1976). From our study and the results reported in the literature, we believe that juvenile nephronophthisis is part of a larger clinicopathologic entity which may be termed the nephronophthisis complex. This includes juvenile nephronophthisis which occurs mainly in children and adolescents.
120,000) exhibits a high affinity to B-galactopyranoside (Drysdale et al. 1968). Cross reaction between these two lectins was excluded by the doubleincubation technique. Moreover, the receptor for PNA is destructible by chymotrypsin, the receptor of RCA by trypsin (Vierbuchen et al. 1980; Klein et al. 1980). The specificity of the lectin binding was proved by inhibition with D-galactose and Blactose for RCA and with asialoglycophorin from human erythrocytes, which contains large amounts of gal-B(1-3) NAcgal (Thomas and Winzler 1969).
Big Bird's Mother Goose by John Barrett